This epilogue presents some concluding thoughts from the author. Increased stochasticity, beginning at the level of the genome, may work its way through the system and eventually translate into aging as a pattern of cells and tissues acting increasingly out of tune. It is now possible to explore a wealth of ever-more sophisticated tools in both cell and molecular biology and the computational sciences to systematically study these cascades of stochastic variation in the various model systems for aging that are now available. This is likely to finally unlock the door to a full understanding of how we grow old.
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